Traumatic intracerebral hemorrhage outcome

• Association Between Cerebral Amyloid Angiopathy and Nontraumatic Subdural Hemorrhage
• Two decades of trends in nontraumatic intracerebral hemorrhage care: A nationwide analysis
• Liberal transfusion strategies reduce sepsis risk and improve neurological recovery in acute brain injury: an updated systematic review and meta-analysis
• Relationship between Tranexamic Acid Use and Safety in Patients with Acute Brain Injury: A Systematic Review and Meta-analysis of Mortality and Thromboembolic Events
• Transfusion Thresholds in Patients With Neurological Injury: Balancing Oxygen Delivery and Risk
• Diagnosis of in-hospital mortality using admission CT perfusion in severe traumatic brain injury patients (ACT-TBI study)
• Advanced lung cancer inflammation index is associated with mortality in critically ill patients with non-traumatic cerebral hemorrhage
• Intrathecal nicardipine for cerebral vasospasm after non-traumatic subarachnoid hemorrhage: a meta-analysis

Cerebral contusions, are frequently associated with surrounding edematous-appearing tissue that exacerbates elevation of intracranial pressure (ICP). Half of all cerebral contusions enlarge in the first hours after injury, with perilesional hypodensity being a significant factor in prediction of expansion ¹⁾.

The presence of Apolipoprotein E, an elevated international normalized ratio, and a higher glucose level (≥ 10 mmol/L) are predictors of Progressive traumatic intracerebral hemorrhage. Additionally, APOE ε4 is not associated with traumatic coagulopathy and patient outcome ²⁾.

A survey of 729 patients with TBI by the TBI European Brain Injury Consortium found that cerebral contusions alone (44%) or in association with subdural haematoma (29%) were the most frequent causes for delayed surgical intervention ³⁾.

The Neutrophil to lymphocyte ratio is easily calculated and might predict the early growth of Traumatic intracerebral hemorrhage for patients suffering from TBI ⁴⁾.

Repeat outpatient CT of asymptomatic patients after nonoperative cerebral contusion and tSAH is very unlikely to demonstrate significant new pathology. Given the cost and radiation exposure associated with CT, imaging should be reserved for patients with significant symptoms or focal findings on neurological examination ⁵⁾.

Intracerebral hemorrhage complications.