An exocytosis is a form of active transport and bulk transport in which a cell transports molecules out of the cell by secreting them through an energy-dependent process.
Ischemic stroke triggers a cascade of events that facilitates neuroprotection and spontaneous recovery, which accounts for a major part of functional recovery. Despite the cellular and molecular facilitations on neural protection, the molecular mechanisms of spontaneous recovery have not been fully understood. Ca2+ -dependent activator protein for secretion 1 (CAPS1), a member of the CAPS family, plays a major role in synaptic transmission and synaptic effectiveness by regulating vesicle exocytosis.