Traumatic [[optic neuropathy]] (TON) is a serious [[complication]] of [[craniofacial]] [[trauma]] that directly or indirectly damages the [[optic nerve]] and can cause severe [[vision loss]]. The [[incidence]] of TON has been gradually increasing in recent years. Research on the protection and [[regeneration]] of the optic nerve after the onset of TON is still at the level of laboratory studies and is insufficient to support the clinical treatment of TON. And, due to without clear [[guideline]]s, there is much ambiguity regarding its diagnosis and management. Clinical interventions for TON include observation only, treatment with [[corticosteroid]]s alone, or [[optic canal decompression]] (with or without steroids). There is controversy in clinical practice concerning which treatment is the best. A review of available studies shows that the [[visual acuity]] of patients with TON can be significantly improved after OC decompression surgery (especially [[endoscopic transnasal transseptal optic canal decompression]] (ETOCD)) with or without the use of [[corticosteroid]]s. And new findings of laboratory studies such as mitochondrial therapy, lipid change studies, and other studies in favor of TON therapy have also been identified
((Chen B, Zhang H, Zhai Q, Li H, Wang C, Wang Y. [[Traumatic optic neuropathy]]: a [[review]] of current studies. Neurosurg Rev. 2022 Jan 16. doi: 10.1007/s10143-021-01717-9. Epub ahead of print. PMID: 35034261.)).